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First PublishedTitleAuthor(s)Type
Oct-2009Diminished hippocalcin expression in Huntington's disease brain does not account for increased striatal neuron vulnerability as assessed in primary neurons.Rudinskiy, N; Kaneko, YA; Beesen, AA; Gokce, O; Régulier, E; Déglon, N; Luthi-Carter, RJournal Article
17-Oct-2008The ratio of monomeric to aggregated forms of Abeta40 and Abeta42 is an important determinant of amyloid-beta aggregation, fibrillogenesis, and toxicity.Jan, A; Gokce, O; Luthi-Carter, R; Lashuel, HAJournal Article
Jan-2009Implication of the JNK pathway in a rat model of Huntington's disease.Perrin, V; Dufour, N; Raoul, C; Hassig, R; Brouillet, E; Aebischer, P; Luthi-Carter, R; Déglon, NJournal Article
Mar-2009Sustained effects of nonallele-specific Huntingtin silencing.Drouet, V; Perrin, V; Hassig, R; Dufour, N; Auregan, G; Alves, S; Bonvento, G; Brouillet, E; Luthi-Carter, R; Hantraye, P; Déglon, NJournal Article
24-Sep-2008Dysregulation of gene expression in primary neuron models of Huntington's disease shows that polyglutamine-related effects on the striatal transcriptome may not be dependent on brain circuitry.Runne, H; Régulier, E; Kuhn, A; Zala, D; Gokce, O; Perrin, V; Sick, B; Aebischer, P; Déglon, N; Luthi-Carter, RJournal Article
Jan-2008Haloperidol protects striatal neurons from dysfunction induced by mutated huntingtin in vivo.Charvin, D; Roze, E; Perrin, V; Deyts, C; Betuing, S; Pagès, C; Régulier, E; Luthi-Carter, R; Brouillet, E; Déglon, N; Caboche, JJournal Article
Apr-2008Comprehensive spatiotemporal transcriptomic analyses of the ganglionic eminences demonstrate the uniqueness of its caudal subdivision.Willi-Monnerat, S; Migliavacca, E; Surdez, D; Delorenzi, M; Luthi-Carter, R; Terskikh, AVJournal Article