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|Title:||Airway hyperresponsiveness is dissociated from airway wall structural remodeling.|
|Citation:||J ALLERGY CLIN IMMUNOL, 2008, 122 (2), pp. 335-3.41E+05|
|Abstract:||Nonasthmatic eosinophilic bronchitis (EB) has emerged as a useful tool to study the structural and inflammatory mechanisms of airway hyperresponsiveness (AHR) in asthma. We have previously shown that vascular remodeling and reticular basement membrane (RBM) thickening are present in EB. However, it is not known whether other features of structural remodeling including increased airway smooth muscle (ASM) mass, matrix deposition, and glandular hyperplasia are also present in EB.|
|Appears in Collections:||Published Articles, Dept. of Infection, Immunity and Inflammation|
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