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|Title:||Turnover of human tubular cells exposed to proteins in vivo and in vitro.|
|Citation:||KIDNEY INT, 2001, 59 (2), pp. 507-514|
|Abstract:||The cause of tubulointerstitial pathology in glomerular disease is uncertain. Proteinuria may be a causative factor and has been shown to increase the turnover of tubular cells in a rat model of proteinuria. We investigated whether increased tubular cell proliferation occurs in human proteinuric renal disease. A human cell culture model was used to investigate the effects of proteins on tubular cell turnover further.|
|Appears in Collections:||Published Articles, Dept. of Infection, Immunity and Inflammation|
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