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Title: Tumour-associated tenascin-C isoforms promote breast cancer cell invasion and growth by matrix metalloproteinase-dependent and independent mechanisms.
Authors: Hancox, RA
Allen, MD
Holliday, DL
Edwards, DR
Pennington, CJ
Guttery, DS
Shaw, JA
Walker, RA
Pringle, JH
Jones, JL
First Published: 2009
Citation: BREAST CANCER RES, 2009, 11 (2), pp. R24-R24
Abstract: The stromal microenvironment has a profound influence on tumour cell behaviour. In tumours, the extracellular matrix (ECM) composition differs from normal tissue and allows novel interactions to influence tumour cell function. The ECM protein tenascin-C (TNC) is frequently up-regulated in breast cancer and we have previously identified two novel isoforms - one containing exon 16 (TNC-16) and one containing exons 14 plus 16 (TNC-14/16).
DOI Link: 10.1186/bcr2251
eISSN: 1465-542X
Type: Journal Article
Appears in Collections:Published Articles, Dept. of Cancer Studies and Molecular Medicine

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