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|Title:||Tumour-associated tenascin-C isoforms promote breast cancer cell invasion and growth by matrix metalloproteinase-dependent and independent mechanisms.|
|Citation:||BREAST CANCER RES, 2009, 11 (2), pp. R24-R24|
|Abstract:||The stromal microenvironment has a profound influence on tumour cell behaviour. In tumours, the extracellular matrix (ECM) composition differs from normal tissue and allows novel interactions to influence tumour cell function. The ECM protein tenascin-C (TNC) is frequently up-regulated in breast cancer and we have previously identified two novel isoforms - one containing exon 16 (TNC-16) and one containing exons 14 plus 16 (TNC-14/16).|
|Appears in Collections:||Published Articles, Dept. of Cancer Studies and Molecular Medicine|
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