Please use this identifier to cite or link to this item: http://hdl.handle.net/2381/17135
Title: Common variants at 10 genomic loci influence hemoglobin A₁(C) levels via glycemic and nonglycemic pathways.
Authors: Soranzo, N.
Sanna, S.
Wheeler, E.
Gieger, C.
Radke, D.
Dupuis, J.
Bouatia-Naji, N.
Langenberg, C.
Prokopenko, I.
Stolerman, E.
Sandhu, M. S.
Oexle, K.
Egan, J. M.
Elosua, R.
Ferrucci, L.
Forouhi, N.
Fox, C. S.
Franklin, C.
Franzosi, M. G.
Gallina, S.
Goel, A.
Devaney, J. M.
Rader, D. J.
Graessler, J.
Olla, N.
Grallert, H.
Greinacher, A.
Hadley, D.
Hall, A.
Hamsten, A.
Hayward, C.
Heath, S.
Schunkert, H.
Herder, C.
Rathmann, W.
Homuth, G.
Hottenga, J. J.
Pankow, J. S.
Hunter-Merrill, R.
Illig, T.
Jackson, A. U.
Jula, A.
Kleber, M.
Schwarz, P.
Knouff, C. W.
Kong, A.
Ripatti, S.
Kooner, J.
Köttgen, A.
Kovacs, P.
Payne, F.
Krohn, K.
Kühnel, B.
Kuusisto, J.
Seedorf, U.
Laakso, M.
Lathrop, M.
Lecoeur, C.
Rocheleau, G.
Li, M.
Li, M.
Loos, R. J.
Luan, J.
Peden, J. F.
Lyssenko, V.
Selvin, E.
Mägi, R.
Magnusson, P. K.
Mälarstig, A.
Mangino, M.
Roden, M.
Martínez-Larrad, M. T.
März, W.
McArdle, W. L.
McPherson, R.
Meisinger, C.
Serrano-Ríos, M.
Pedersen, N. L.
Meitinger, T.
Melander, O.
Mohlke, K. L.
Mooser, V. E.
Rudan, I.
Morken, M. A.
Narisu, N.
Nathan, D. M.
Nauck, M.
Shrader, P.
Peltonen, L.
Perola, M.
Polasek, O.
Heeney, M. M.
Salomaa, V.
Saxena, R.
Schlessinger, D.
Barnes, D.
Silveira, A.
Siscovick, D.
Song, K.
Campbell, H.
Boerwinkle, E.
Spector, T. D.
Reilly, M. P.
Stefansson, K
Steinthorsdottir, V.
Strachan, D. P.
Strawbridge, R.
Stumvoll, M.
Surakka, I.
Swift, A. J.
Cao, A.
Tanaka, T.
Böhm, B.
Teumer, A.
Thorleifsson, G.
Ricketts, S. L.
Thorsteinsdottir, U.
Tönjes, A.
Usala, G.
Vitart, V.
Völzke, H.
Chambers, J.
Wallaschofski, H.
Waterworth, D. M.
Bonnefond, A.
Watkins, H.
Wichmann, H. E.
Wild, S. H.
Stewart, A. F.
Willemsen, G.
Williams, G. H.
Wilson, J. F.
Clark, R.
Winkelmann, J.
Wright, A. F.
WTCCC
Bonnycastle, L. L.
Zabena, C.
Zhao, J. H.
Epstein, S. E.
Erdmann, J.
Voight, B. F.
Hakonarson, H. H.
Collins, F. S.
Kathiresan, S.
Khaw, K. T.
Roberts, R.
Samani, N. J.
Boomsma, D. I.
Fleming, M. D.
Sladek, R.
Abecasis, G.
Boehnke, M.
Froguel, P.
Coresh, J.
Willenborg, C.
Groop, L.
McCarthy, M. I.
Kao, W. H.
Florez, J. C.
Bornstein, S. R.
Uda, M.
Wareham, N. J.
Barroso, I.
Meigs, J. B.
de Geus, E. J.
Wright, B.
Altshuler, D.
Arking, D.
Balkau, B.
Böttcher, Y.
Bumpstead, S.
Burnett-Miller, M. S.
O'Donnell, C.
Dei, M.
Deloukas, P.
Döring, A.
Porcu, E.
First Published: Dec-2010
Citation: Diabetes, 2010, 59 (12), pp. 3229-3239
Abstract: Glycated hemoglobin (HbA₁(c)), used to monitor and diagnose diabetes, is influenced by average glycemia over a 2- to 3-month period. Genetic factors affecting expression, turnover, and abnormal glycation of hemoglobin could also be associated with increased levels of HbA₁(c). We aimed to identify such genetic factors and investigate the extent to which they influence diabetes classification based on HbA₁(c) levels.
DOI Link: 10.2337/db10-0502
eISSN: 1939-327X
Links: http://hdl.handle.net/2381/17135
http://diabetes.diabetesjournals.org/content/59/12/3229
Type: Journal Article
Rights: © 2010 by the American Diabetes Association. Readers may use this article as long as the work is properly cited, the use is educational and not for profit, and the work is not altered. See http://creativecommons.org/licenses/by -nc-nd/3.0/ for details.
Appears in Collections:Published Articles, Dept. of Cardiovascular Sciences

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