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|Title:||Molecular changes in extracellular matrix turnover after renal ischaemia-reperfusion injury.|
|Citation:||BR J SURG, 2000, 87 (9), pp. 1188-1192|
|Abstract:||Renal ischaemia-reperfusion (IR) injury is an inevitable consequence of transplantation and contributes to later graft fibrosis. This study aimed to elucidate the possible mechanisms by studying the expression of genes associated with extracellular matrix (ECM) turnover.|
|Appears in Collections:||Published Articles, Dept. of Infection, Immunity and Inflammation|
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