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|Title:||The experimental agent pirfenidone reduces pro-fibrotic gene expression in a model of tacrolimus-induced nephrotoxicity.|
|Citation:||J SURG RES, 2005, 125 (2), pp. 137-143|
|Abstract:||Tacrolimus nephrotoxicity is thought to contribute to renal allograft dysfunction and subsequent failure, a process that is underpinned by alterations in mRNA expression of genes involved in matrix metabolism. The new anti-fibrotic pirfenidone was tested for its potential to reverse markers of renal dysfunction.|
|Appears in Collections:||Published Articles, Dept. of Infection, Immunity and Inflammation|
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