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|Title:||A biphasic response to nitric oxide donation in an ex vivo model of donation after cardiac death renal transplantation.|
|Citation:||J SURG RES, 2012, 175 (2), pp. 316-321|
|Abstract:||Donation after cardiac death (DCD) donors are vital to maximize the organ donor pool. Reperfusion injury (RI) is an important sequela in DCD organs due to warm and cold ischemia. RI manifests clinically as a high incidence of delayed graft function (DGF) and primary non-function (PNF) compared with donation after brain death organs. The importance of nitric oxide (NO) in the generation of reperfusion injury is pivotal.|
|Appears in Collections:||Published Articles, Dept. of Infection, Immunity and Inflammation|
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