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|Title:||Nitric oxide generation is increased in experimental renal warm ischaemia-reperfusion injury.|
|Citation:||BR J SURG, 1998, 85 (12), pp. 1663-1668|
|Abstract:||Nitric oxide has a clearly defined place in normal renal homoeostasis while there is a continuing debate as to its role under pathophysiological conditions. This study investigated the role of nitric oxide in a model of renal warm ischaemia-reperfusion injury.|
|Appears in Collections:||Published Articles, Dept. of Infection, Immunity and Inflammation|
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