Please use this identifier to cite or link to this item: http://hdl.handle.net/2381/8033
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dc.contributor.authorHollox, Edward J.-
dc.date.accessioned2010-06-14T10:15:14Z-
dc.date.available2010-06-14T10:15:14Z-
dc.date.issued2009-
dc.identifier.citationAmerican Journal Gastroenterology, 2009, 105(2), pp.360–362en_GB
dc.identifier.issn0002-9270-
dc.identifier.issn1572-0241-
dc.identifier.urihttp://www.nature.com/ajg/journal/v105/n2/full/ajg2009573a.htmlen_GB
dc.identifier.urihttp://hdl.handle.net/2381/8033-
dc.description.abstractRecent research has suggested that a family of antimicrobial and inflammatory molecules, called β-defensins, are involved in the etiology of Crohn's disease. In this issue, Bentley and colleagues provide data that disagree with previous studies. They show that the β-defensins that are copy number polymorphic have, on average, higher genomic copy number in patients when compared with healthy controls. This editorial places these new data in the context of previous research on the genetics of β-defensin copy number polymorphism and association with Crohn's disease. It also suggests a path allowing this research area to move forward with confidence.en_GB
dc.language.isoenen_GB
dc.publisherNature Publishing Groupen_GB
dc.rightsThis is the author's final draft of the paper published as American Journal Gastroenterology, 2009, 105(2), pp.360–362 The final published version is available at http://www.nature.com/ajg/journal/v105/n2/abs/ajg2009573a.html. doi:10.1038/ajg.2009.573.-
dc.titleß-defensins and Crohn's disease: confusion from counting copiesen_GB
dc.typeArticleen_GB
dc.identifier.doi10.1038/ajg.2009.573-
Appears in Collections:Published Articles, Dept. of Genetics

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