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First PublishedTitleAuthor(s)Type
Oct-2009Diminished hippocalcin expression in Huntington's disease brain does not account for increased striatal neuron vulnerability as assessed in primary neurons.Rudinskiy, N; Kaneko, YA; Beesen, AA; Gokce, O; Régulier, E; Déglon, N; Luthi-Carter, RJournal Article
1-Aug-2007Mutant huntingtin's effects on striatal gene expression in mice recapitulate changes observed in human Huntington's disease brain and do not differ with mutant huntingtin length or wild-type huntingtin dosage.Kuhn, A; Goldstein, DR; Hodges, A; Strand, AD; Sengstag, T; Kooperberg, C; Becanovic, K; Pouladi, MA; Sathasivam, K; Cha, JH; Hannan, AJ; Hayden, MR; Leavitt, BR; Dunnett, SB; Ferrante, RJ; Albin, R; Shelbourne, P; Delorenzi, M; Augood, SJ; Faull, RL; Olson, JM; Bates, GP; Jones, L; Luthi-Carter, RJournal Article
Feb-2006Capucin: a novel striatal marker down-regulated in rodent models of Huntington disease.de Chaldée M; Brochier, C; Van de Vel A; Caudy, N; Luthi-Carter, R; Gaillard, MC; Elalouf, JMJournal Article
15-May-2007Modulation of nucleosome dynamics in Huntington's disease.Stack, EC; Del Signore SJ; Luthi-Carter, R; Soh, BY; Goldstein, DR; Matson, S; Goodrich, S; Markey, AL; Cormier, K; Hagerty, SW; Smith, K; Ryu, H; Ferrante, RJJournal Article
17-Oct-2008The ratio of monomeric to aggregated forms of Abeta40 and Abeta42 is an important determinant of amyloid-beta aggregation, fibrillogenesis, and toxicity.Jan, A; Gokce, O; Luthi-Carter, R; Lashuel, HAJournal Article
Jan-2009Implication of the JNK pathway in a rat model of Huntington's disease.Perrin, V; Dufour, N; Raoul, C; Hassig, R; Brouillet, E; Aebischer, P; Luthi-Carter, R; Déglon, NJournal Article
Mar-2009Sustained effects of nonallele-specific Huntingtin silencing.Drouet, V; Perrin, V; Hassig, R; Dufour, N; Auregan, G; Alves, S; Bonvento, G; Brouillet, E; Luthi-Carter, R; Hantraye, P; Déglon, NJournal Article
Dec-2008Biological and potential therapeutic roles of sirtuin deacetylases.Taylor, DM; Maxwell, MM; Luthi-Carter, R; Kazantsev, AGJournal Article
24-Sep-2008Dysregulation of gene expression in primary neuron models of Huntington's disease shows that polyglutamine-related effects on the striatal transcriptome may not be dependent on brain circuitry.Runne, H; Régulier, E; Kuhn, A; Zala, D; Gokce, O; Perrin, V; Sick, B; Aebischer, P; Déglon, N; Luthi-Carter, RJournal Article